Savannah Tallino

PhD Student - Mentor: Ramon Velazquez, PhD, Interdisciplinary Graduate Program in Neuroscience Students

Graduate Research Assistant, ASU-Banner Neurodegenerative Disease Research Center

savannah.tallino@asu.edu

Biodesign Building C 1001 S McAllister Ave Tempe, AZ 85281
Mail code: 7601

Campus: Tempe
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Student Information

Graduate Student
Neuroscience
The College of Lib Arts & Sci

Long Bio

Born and raised in rural Alaska, Savannah attended the University of Alaska Anchorage (UAA) for both her BS (Natural Science) and MS (Biology), the latter under Dr. Jason Burkhead working on the role of dietary copper in non-alcoholic fatty liver disease. She also served as an adjunct instructor and then term instructor for several years in UAA's Biological Sciences department, teaching laboratory and lecture courses including non-majors introductory biology, general biology, cell biology, genetics, and human anatomy and physiology.

Savannah first joined the ASU community in ASU's School of Life Sciences (SOLS) as a grant administrator before switching back to research. She now works within Biodesign's ASU-Banner Neurodegenerative Disease Research Center as member of the Velazquez Lab, where she is involved in projects investigating mechanisms of basal forebrain cholinergic neuron alterations in mouse models of Down Syndrome and their relationship to neurodegeneration in Alzheimer's disease. She began the SOLS Interdisciplinary Graduate Program in Neuroscience PhD program in Fall 2021.

Education

Arizona State University - PhD student, SOLS Interdisciplinary Graduate Program in Neuroscience (Aug 2021-Present)

University of Alaska Anchorage - M.S. in Biological Sciences, Aug 2014

University of Alaska Anchorage - B.S. in Natural Sciences, Dec 2010. Cum Laude.

Publications

Dave, N.*, Judd, J.*, Decker, A., Winslow, W., Sarette, P., Espinosa, O., Tallino, S., Bartholomew, S., Bilal, A., Sandler, J., McDonough, I., Winstone, J., Blackwood, E., Glembotski, C., Karr, T., Velazquez, R. (2023). Dietary choline intake is necessary to prevent systems-wide organ pathology and reduce Alzheimer’s disease hallmarks. Aging Cell 22(2):e13775. (*Co-Authors)

Tallino, S., Winslow, W., Velazquez, R. (2023). Trajectory of amyloid pathology in a Down syndrome model. Aging 15(2):295-297.

Tallino, S., Winslow, W., Bartholomew, S., Velazquez, R. (2022). Temporal and brain region specific elevations of soluble Amyloid Beta40-42 in the Ts65Dn mouse model of Down syndrome and Alzheimer's disease. Aging Cell e13590.

Winslow, W., Mcdonough, I., Tallino, S., Decker, A., Vural, A., Velazquez, R. (2021). IntelliCage automated behavioral phenotyping reveals behavior deficits in the 3xTg-AD mouse model of Alzheimer's disease associated with brain weight. Frontiers in Aging Neuroscience 13:720214.

Mifflin, M., Winslow, W., Surendra, L., Tallino, S., Vural, A., and Velazquez, R. (2021). Sex differences in the IntelliCage and the Morris water maze in the APP/PS1 mouse model of amyloidosis. Neurobiology of Aging 101:130-140.

Morrell, A., Tallino, S., Yu, L., and Burkhead, J. L. (2017). The role of insufficient copper in lipid synthesis and fatty-liver disease. IUBMB Life 69(4):263-270.

Tallino, S., Duffy, M., Ralle, M., Cortés, M. P., Latorre, M., and Burkhead, J. L. (2015). Nutrigenomics analysis reveals that copper deficiency and dietary sucrose up-regulate inflammation, fibrosis, and lipogenic pathways in a mature rat model of nonalcoholic fatty liver disease. Journal of Nutritional Biochemistry 26(10):996-1006.

Tallino, S. (2014). Copper deficiency and sucrose consumption: Factors in the “western diet” and their relationship to non-alcoholic fatty liver disease and cognitive decline (Order No. 1563531). Available from ProQuest Dissertations and Theses Global. (1612508555).

Expertise Areas